Finding fibroblast growth factor 19 during cholestasis: Does x mark the spot?
Author(s): ,
Rowan F. van Golen
Affiliations:
Department of Gastroenterology and Hepatology, Leiden University Medical Center, Leiden, the Netherlands
Corresponding author. Address: Leiden University Medical Center, Leiden University, Albinusdreef 2, 2333 ZA Leiden, the Netherlands.
Lianne R. de Haan
Affiliations:
Department of Surgery, Surgical Laboratory, Academic Medical Center, Amsterdam, the Netherlands
EASL LiverTree™. van Golen R. Dec 1, 2018; 256783
Rowan van Golen
Rowan van Golen

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[1]. L.R. De Haan - Post-hepatectomy liver regeneration in the context of bile acid homeostasis and the gut-liver signaling axis
[2]. F.G. Schaap - High expression of the bile salt-homeostatic hormone fibroblast growth factor 19 in the liver of patients with extrahepatic cholestasis
[3]. K. Brandl - Dysregulation of serum bile acids and FGF19 in alcoholic hepatitis
[4]. E. Wunsch - Expression of hepatic fibroblast growth factor 19 is enhanced in primary biliary cirrhosis and correlates with severity of the disease
[5]. R.F. van Golen - The pathophysiology of human obstructive cholestasis is mimicked in cholestatic gold syrian hamsters
[6]. N. Ijssennagger - Gene expression profiling in human precision cut liver slices upon treatment with the FXR agonist obeticholic acid
[7]. K.H. Song - Bile acids activate fibroblast growth factor 19 signaling in human hepatocytes to inhibit cholesterol 7alpha-hydroxylase gene expression
[8]. W.E. Naugler - Fibroblast growth factor signaling controls liver size in mice with humanized livers
[9]. S.J. Zweers - The human gallbladder secretes fibroblast growth factor 19 into bile: Towards defining the role of fibroblast growth factor 19 in the enterobiliary tract
[10]. P. Hartmann - Modulation of the intestinal bile acid/farnesoid X receptor/fibroblast growth factor 15 axis improves alcoholic liver disease in mice
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